Yes, genetic hair loss, also known as androgenetic alopecia or male-pattern baldness, is common in men. It is the most prevalent cause of hair loss and affects a significant percentage of the male population. The condition is influenced by genetic and hormonal factors.
The inheritance of androgenetic alopecia is believed to involve a combination of genetic factors from both sides of the family. The condition is linked to the hormone dihydrotestosterone (DHT), which is a byproduct of testosterone. DHT affects hair follicles, leading to a gradual shrinking of the follicles and a shortening of the hair growth cycle. Over time, this results in finer and shorter hair until the follicles become inactive and no longer produce visible hair.
The pattern of male-pattern baldness typically follows a specific progression:
- Receding Hairline: The hairline at the temples begins to recede, forming an “M” shape.
- Thinning at the Crown: Hair loss progresses at the crown of the head, leading to increased baldness in this area.
- Thinning and Balding Continue: The hairline recedes further, and the crown becomes more pronounced, often leaving hair at the sides and back of the head.
The onset and progression of male-pattern baldness can vary among individuals, and some men may experience more significant hair loss than others. The condition can start as early as the late teens or early twenties, and its prevalence increases with age.
While genetic hair loss is common, there are various treatment options available, including medications such as minoxidil and finasteride, as well as surgical interventions like hair transplant surgery. These treatments can help slow down hair loss, promote hair regrowth, or restore hair density, depending on the individual’s response to the treatment. It’s advisable for individuals experiencing hair loss to consult with a Hair Restoration Center Patient Advisor to discuss the most appropriate options based on their specific situation. Schedule a complimentary consultation today or call 1 (888) 589-3064.
